パスウェイパネル
Reactomeの生物学的パスウェイごとにqRT-PCRプライマーを探せます。
1862 件のパネル
- Synthesis of PG 8 遺伝子
- Synthesis of UDP-N-acetyl-glucosamine 8 遺伝子
- Synthesis of diphthamide-EEF2 8 遺伝子
- Synthesis of epoxy (EET) and dihydroxyeicosatrienoic acids (DHET) 8 遺伝子
- TGFBR3 regulates TGF-beta signaling 8 遺伝子
- TRAIL signaling 8 遺伝子
- Terminal pathway of complement 8 遺伝子
- The retinoid cycle in cones (daylight vision) 8 遺伝子
- Transport of RCbl within the body 8 遺伝子
- VEGF binds to VEGFR leading to receptor dimerization 8 遺伝子
- Vitamin C (ascorbate) metabolism 8 遺伝子
- WNT mediated activation of DVL 8 遺伝子
- Zymostenol biosynthesis via lathosterol (Kandutsch-Russell pathway) 8 遺伝子
- 2-LTR circle formation 7 遺伝子
- APEX1-Independent Resolution of AP Sites via the Single Nucleotide Replacement Pathway 7 遺伝子
- ARMS-mediated activation 7 遺伝子
- Activated NTRK2 signals through PI3K 7 遺伝子
- Activation of NIMA Kinases NEK9, NEK6, NEK7 7 遺伝子
- Activation of the mRNA upon binding of the cap-binding complex and eIFs, and subsequent binding to 43S 7 遺伝子
- Acyl chain remodeling of DAG and TAG 7 遺伝子
- Amine ligand-binding receptors 7 遺伝子
- Aryl hydrocarbon receptor signalling 7 遺伝子
- Attachment of GPI anchor to uPAR 7 遺伝子
- CDH11 homotypic and heterotypic interactions 7 遺伝子
- CREB phosphorylation 7 遺伝子
- Calmodulin induced events 7 遺伝子
- Cation-coupled Chloride cotransporters 7 遺伝子
- Cobalamin (Cbl) metabolism 7 遺伝子
- Coenzyme A biosynthesis 7 遺伝子
- Constitutive Signaling by NOTCH1 t(7;9)(NOTCH1:M1580_K2555) Translocation Mutant 7 遺伝子
- DAG and IP3 signaling 7 遺伝子
- DEx/H-box helicases activate type I IFN and inflammatory cytokines production 7 遺伝子
- Defective CSF2RA causes SMDP4 7 遺伝子
- Defective CSF2RB causes SMDP5 7 遺伝子
- Defective HLCS causes multiple carboxylase deficiency 7 遺伝子
- Defective binding of VWF variant to GPIb:IX:V 7 遺伝子
- Digestion of dietary lipid 7 遺伝子
- Enhanced binding of GP1BA variant to VWF multimer:collagen 7 遺伝子
- Erythropoietin activates Phospholipase C gamma (PLCG) 7 遺伝子
- Erythropoietin activates STAT5 7 遺伝子
パスウェイデータの出典：Reactome（CC0）。